Although it is valuable to detect coronary artery occlusion before sudden death, angina or heart attack, ideally, you should try to prevent blockage formation. This is why it is important for you to understand how atherosclerosis develops and what factors accelerate the development of atherosclerosis. Even if you already have coronary heart disease and have undergone coronary artery bypass surgery, you will want to reduce the likelihood of blockage.
Coronary atherosclerosis gradually progresses to coronary heart disease. The term atherosclerosis comes from Greek atherosclerosis, meaning porridge and hard bones, meaning hard. At birth, our coronary arteries are fully open, free from obstruction, and blood flow is not compromised. Between 10 and 20 years old, small lipid deposits called "fat stripes" begin to appear in the inner lining of the coronary arteries. Over time, some fat strips gradually become larger deposits called "fibrous plaques." As fibrous plaque forms, it protrudes into the opening of the coronary arteries.
These early stages of atherosclerosis progressed slowly during adolescence and in the twenties and thirties, but by the age of 45 or 50, atherosclerosis is more severe in many people in our society and may cause coronary heart disease. If you have a risk factor or a hereditary genetic problem to treat body fat, you are more likely to develop atherosclerosis. The same factors may accelerate the early deposition of fatty streak in your child's coronary arteries.
Atherosclerosis has plagued many people throughout human history. For example, it was found in Egyptian mummies, but not all mammals are not susceptible to the disease. Rats and dogs are very resistant, at least in part because most of their blood cholesterol contains high-density lipoprotein (HDL), or "good" cholesterol. In contrast, humans transport cholesterol in most of the blood to atherosclerotic low-density lipoprotein (LDL). When animals eat a diet rich in cholesterol or saturated fat, those that cannot tolerate, such as some non-human primates and rabbits, develop atherosclerosis; those who are resistant, such as dogs and rats, do not.
In 1908, Russian pathologist Ignatowsky first observed the relationship between dietary cholesterol and atherosclerosis in rabbits. Later research on non-human primates confirmed the direct relationship between dietary cholesterol and saturated fat, cholesterol levels in the blood, and the development of atherosclerosis.
Will the process of atherosclerosis reverse?
The answer is yes. Studies have shown that when cholesterol levels in non-human primate blood are reduced by diet or drugs, the deposition of atherosclerosis in their coronary arteries becomes smaller. A study also shows that this is correct in humans.
In the 1960s, medical researchers determined that not all countries have the same number of atherosclerosis in their population. The International Coronary Heart Disease and Atherosclerosis Project studied people in 14 different countries. The arteries of 22,509 dead people between the ages of 10 and 69 were examined under a microscope. In his book "Geographical Pathology of Coronary Heart Disease and Atherosclerosis DR," Henry McGill ranks among four populations based on fat intake, blood cholesterol levels, and coronary heart disease and atherosclerosis. twelve. He found a direct and very important relationship between these factors; in areas with the highest levels of coronary heart disease and atherosclerosis, such as the United States and Norway, the average blood cholesterol level was significantly higher and fat consumption was significantly increased.
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Orignal From: Coronary heart disease and atherosclerosis
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